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Clinical Policy Bulletin:
Voice Therapy
Number: 0646


Policy

Note: Voice therapy is subject to any benefit limitations and exclusions applicable to speech therapy. See CPB 243 - Speech Therapy.

  1. Aetna considers voice therapy medically necessary to restore the ability of the member to produce speech sounds from the larynx for any of the following indications:

    1. Vocal cord paralysis; or
    2. Vocal cord nodules; or
    3. Following surgery or traumatic injury to the vocal cords; or
    4. Spastic (spasmodic) dysphonia; or
    5. Following treatment for laryngeal (glottic) carcinoma.

  2. Aetna considers voice therapy not medically necessary for any of the following indications:

    1. Laryngitis; or
    2. Occupational or recreational purposes (e.g., public speaking, singing, etc.); or
    3. Improvement of voice quality; or
    4. Muscle tension dysphonia (functional dysphonia); or
    5. Essential voice tremor.

Note: Megaphones or amplifiers (e.g., ChatterVox, Mega Mite Megaphone) may be of use in the absence of illness or injury and therefore do not meet Aetna's definition of covered durable medical equipment.

Note: An electronic artificial larynx (artificial voice box) that is used by laryngectomized individuals and persons with a permanently inoperative larynx is covered as a prosthetic. See note regarding electronic speech aids accompanying CPB 437 - Speech Generating Devices. See also  CPB 560 - Voice Prosthesis for Voice Rehabilitation Following Total Laryngectomy.



Background

Vocal cord paralysis is a voice disorder that occurs when one or both of the vocal cords (or vocal folds) do not open or close properly (NIDCD, 1999). Vocal cord paralysis is a common disorder, and symptoms can range from mild to life threatening. Someone who has vocal cord paralysis often has difficulty swallowing and coughing because food or liquids slip into the trachea and lungs. This happens because the paralyzed cord or cords remain open, leaving the airway passage and the lungs unprotected.

Vocal cord paralysis may be caused by head trauma, a neurological insult such as a stroke, a neck injury, lung or thyroid cancer, a tumor pressing on a nerve, or a viral infection (NIDCD, 1999). In older people, vocal cord paralysis is a common problem affecting voice production. People with certain neurological conditions, such as multiple sclerosis or Parkinson's disease, or people who have had a stroke may experience vocal cord paralysis. In many cases, however, the cause is unknown.

People who have vocal cord paralysis experience abnormal voice changes, changes in voice quality, and discomfort from vocal straining (NIDCD, 1999). For example, if only one vocal cord is damaged, the voice is usually hoarse or breathy. Changes in voice quality, such as loss of volume or pitch, may also be noticeable. Damage to both vocal cords, although rare, usually causes people to have difficulty breathing because the air passage to the trachea is blocked.

Vocal cord paralysis is usually diagnosed by an otolaryngologist (NIDCD, 1999). Noting the symptoms the patient has experienced, the otolaryngologist will ask how and when the voice problems started in order to help determine their cause. Next, the otolaryngologist listens carefully to the patient's voice to identify breathiness or harshness. Then, using an endoscope, the otolaryngologist looks directly into the throat at the vocal cords. A speech-language pathologist may also use an acoustic spectrograph, an instrument that measures voice frequency and clarity, to study the patient's voice and document its strengths and weaknesses.

There are several methods for treating vocal cord paralysis, among them surgery and voice therapy. In some cases, the voice returns without treatment during the first year after damage (NIDCD, 1999). For that reason, doctors often delay corrective surgery for at least a year to be sure the voice does not recover spontaneously. During this time, the suggested treatment is usually voice therapy, which may involve exercises to strengthen the vocal cords or improve breath control during speech. Sometimes, a speech-language pathologist must teach patients to talk in different ways. For instance, the therapist might suggest that the patient speak more slowly or consciously open the mouth wider when speaking.

Surgery involves adding bulk to the paralyzed vocal cord or changing its position (NIDCD, 1999). To add bulk, an otolaryngologist injects a substance, commonly Teflon, into the paralyzed cord. Other substances currently used are collagen, silicone, and body fat. The added bulk reduces the space between the vocal cords so the non-paralyzed cord can make closer contact with the paralyzed cord and thus improve the voice.

Sometimes an operation that permanently shifts a paralyzed cord closer to the center of the airway may improve the voice (NIDCD, 1999). Again, this operation allows the non-paralyzed cord to make better contact with the paralyzed cord. Adding bulk to the vocal cord or shifting its position can improve both voice and swallowing. After these operations, patients may also undergo voice therapy, which often helps to fine-tune the voice.

Treating people who have two paralyzed vocal cords may involve performing a surgical procedure called a tracheotomy to help breathing (NIDCD, 1999). In a tracheotomy, an incision is made in the front of the patient's neck and a breathing tube (tracheotomy tube) is inserted through a hole, called a stoma, into the trachea. Rather than breathing through the nose and mouth, the patient now breathes through the tube. Following surgery, the patient may need therapy with a speech-language pathologist to learn how to care for the breathing tube properly and how to reuse the voice.

Vocal cord nodules (singer's nodules) are small, hard, callus like growths that usually appear singly on the vocal cord (Merck, 1997). Nodules consist of condensations of hyaline connective tissue in the lamina propria at the junction of the anterior 1/3 and posterior 2/3 of the free edges of the true vocal cords. Vocal cord polyps are small, soft growths that usually appear singly on a vocal cord. They are most often caused by vocal abuse or long-term exposure to irritants, such as chemical fumes or cigarette smoke.

Vocal cord nodules are caused by chronic voice abuse, such as yelling, shouting, or using an unnaturally low frequency (Merck, 1997). Chronic infections caused by allergies and inhalation of irritants, such as cigarette smoke, may also produce these lesions. Hoarseness and a breathy voice result. Carcinoma should be excluded by biopsy.

Treatment for nodules that do not resolve with voice therapy involves surgical removal of the nodules at direct laryngoscopy and correction of the underlying voice abuse. Vocal nodules in children usually regress with voice therapy alone.

If nothing is done to change vocal abuse habits, vocal cord nodules can last a lifetime, and may even recur after surgical removal. With proper voice training with a certified therapist, nodules can disappear with 6 to 10 voice therapy sessions over six to 12 weeks. With rest, some vocal cord polyps will go away on their own within a few weeks. Most, however, will require surgical removal.

Speech therapists use a variety of techniques to restore a patient's ability to produce speech, including:

  • Patients are instructed in voice modification and relative voice rest. At its most extreme, relative voice rest involves an initial period of between 4 and 7 days using the voice no more than 15 minutes in each 24-hour period. It is normally undertaken with speech therapist supervision, once its advantages and disadvantages have been explored with the patient.
  • Patients are instructed to minimizing voice use. This involves speaking no more than is absolutely necessary. Patients are taught to be succinct when speaking, to avoid any loud use of the voice, to keep phone calls brief and to avoid all non-speech voice use (throat clearing, coughing, “voiced” sneezing, crying, “voiced” laughing, and odd sound-effects).
  • Patients are told to avoid any “voice abuse” practices identified by the speech therapist. Patients are instructed to avoid or modify internal (bodily and psychological) and external (environmental) voice-damaging environments as much as possible.
  • Patients are instructed in how to engage in a short warm-up period of controlled, soft vocal exercises before using the voice. Patients may also be advised, where appropriate, to keep a supply of drinking water handy, and to massage under their chin if their mouth becomes dry.
  • Patients are taught good voice production techniques. Patients may be instructed in optimal breathing patterns, to speak more slowly, to articulate clearly, to speak at a comfortable pitch and loudness level, to use pitch change rather than volume change for emphasis, to monitor their posture, to avoid monotone delivery, and to be aware of muscle tension.
  • Patients may be advised to avoid speaking when they are ill or tired. Dehydration, fatigue and other general medical conditions have an effect on the mucosa covering the vocal cords, potentially altering lubrication and vocal efficiency.

Laryngitis due to viral infection usually resolves within one to three weeks. Laryngitis due to vocal abuse will generally go away on its own in a few days with voice rest.

Functional voice disorders are characterized by the presence of vocal symptoms without anatomical laryngeal abnormality. Muscle tension dysphonia (MTD) is the most common disorder in this category. Sama et al (2001) stated that the laryngoscopy features commonly associated with FD are frequently prevalent in the non-dysphonic population and fail to distinguish patients with FD from normal subjects. In regards to a multi-dimensional protocol for assessing functional results of voice therapy, Dejonckere (2000) noted that results show that there is a large variation in the inter-individual and inter-dimensional results of the voice therapy (4 to 26 sessions) -- in the same patient, one dimension may be significantly improved while another one is significantly worsened. Furthermore, Roy (2003) stated that while voice therapy by an experienced speech-language pathologist remains an effective short-term treatment for FD in the majority of cases, but less is known regarding the long-term fate of such intervention. Thus, there is insufficient evidence regarding the effectiveness (especially long-term outcomes) of voice therapy for the treatment of muscle tension dysphonia.

Voice therapy has been shown to be effective in rehabilitating persons treated for early glottic carcinoma. In a randomized, controlled trial, van Gogh and colleagues (2006) evaluated the effectiveness of voice therapy in patients who experienced voice problems after receiving treatments for early glottic cancer.  Of 177 patients, 6 to 120 months after treatment for early glottic carcinoma, 70 patients (40 %) suffered from voice impairment based on a 5-item screening questionnaire.  About 60% of those 70 patients were not interested in participating in the study.  A total of 23 patients who were willing to participate were randomly assigned either to a voice therapy group (n = 12) or to a control group (n = 11).  Multi-dimensional voice analyses (the self-reported Voice Handicap Index [VHI], acoustic and perceptual voice quality analysis, videolaryngostroboscopy, and the Voice Range Profile) were conducted twice: before and after voice therapy or with 3 months in between for the control group.  Statistical analyses of the difference in scores (post-measurement minus pre-measurement) showed significant voice improvement after voice therapy on the total VHI score, percent jitter, and noise-to-harmonics ratio in the voice signal and on the perceptual rating of vocal fry.  The authors concluded that voice therapy proved to be effective in patients who had voice problems after treatment for early glottic carcinoma.  Improvement not only was noticed by the patients (VHI) but also was confirmed by objective voice parameters.

 
CPT Codes / HCPCS Codes / ICD-9 Codes
CPT codes covered if selection criteria are met:
92507
92508
Other CPT codes related to the CPB:
31510 - 31515, 31527 - 31571, 31576 - 31578, 31580 - 31590
31611
92506
92597
HCPCS codes covered if selection criteria are met:
G0153 Services of speech and language pathologist in home health setting, each 15 minutes
S9128 Speech therapy, in the home, per diem
HCPCS codes not covered for indications listed in the CPB:
L8510 Voice amplifier
Other HCPCS codes related to the CPB:
L8500 Artificial larynx, any type
L8505 Artificial larynx replacement battery / accessory, any type
L8507 Tracheo-esophageal voice prosthesis, patient inserted, any type, each
L8509 Tracheo-esophageal voice prosthesis, inserted by a licensed health care provider, any type
V5362 Speech screening
V5363 Language screening
ICD-9 codes covered if selection criteria are met:
478.30 - 478.34 Paralysis of vocal cords or larynx
478.4 Polyp of vocal cord or larynx
478.5 Other diseases of vocal cords
478.79 Other diseases of larynx, not elsewhere classified
V10.21 Personal history of malignant neoplasm of larynx
ICD-9 codes not covered for indications listed in the CPB:
012.3 Tuberculous laryngitis
032.3 Laryngeal diphtheria
034.0 Streptococcal sore throat
095.8 Other specified forms of late symptomatic syphilis
101 Vincent's angina
300.11 Conversion disorder [functional dysphonia]
333.1 Essential and other specified forms of tremor [voice]
438.10 - 438.19 Late effects of cerebrovascular disease, speech and language deficits
464.00 - 464.01 Acute laryngitis
464.20 - 464.21 Acute laryngotracheitis
465.0 Acute laryngopharyngitis
487.1 Influenza with other respiratory manifestations
784.40 - 784.49 Voice disturbance
784.5 Other speech disturbance
V41.4 Problems with voice production
V57.21 Encounter for occupational therapy
Other ICD-9 codes related to the CPB:
161.0 Malignant neoplasm of glottis
161.1 Malignant neoplasm of supraglottis
162.0 - 162.9 Malignant neoplasm of trachea, bronchus, and lung
193 Malignant neoplasm of thyroid gland
197.0 Secondary malignant neoplasm of lung
212.1 Benign neoplasm of larynx
231.0 Carcinoma in situ of larynx
231.2 Carcinoma in situ of respiratory system
332.0 Paralysis agitans
332.1 Secondary Parkinsonism
340 Multiple sclerosis
874.0 - 874.9 Open wound of neck
906.0 Late effect of open wound of head, neck, and trunk
907.1 Late effect of injury to cranial nerve
925.2 Crushing injury of neck
951.7 Injury to hypoglossal nerve
951.8 Injury to other specified cranial nerves
959.09 Injury of face and neck
V10.02 Personal history of malignant neoplasm of other and unspecified oral cavity and pharynx


The above policy is based on the following references:
  1. National Institutes of Health, National Institute on Deafness and Other Communication Disorders (NIDCD). Vocal cord paralysis. NIDCD Health Information. Bethesda, MD: NIDCD; June 1999. Available at: http://www.nidcd.nih.gov/health/voice/vocalparal.asp. Accessed July 26, 2002.
  2. National Jewish Medical and Research Center. Vocal cord dysfunction. Medfacts. Denver, CO: National Jewish; updated July 23, 2002. Available at: http://www.nationaljewish.org/medfacts/vocal.html. Accessed July 26, 2002.
  3. Pedersen M, McGlashan J. Surgical versus non-surgical interventions for vocal cord nodules. Cochrane Database Syst Rev. 2001;(2):CD001934.
  4. Merck & Co., Inc. Vocal cord nodules (singer's nodules). In: Merck Manual of Diagnosis and Therapy. 17th ed. MH Beers, R Berkow, eds. White House Station, NJ: Merck; 1997.
  5. Ahern R, Lippincott LH, Wisdom G. Voice rehabilitation after laryngectomy: An overview. J La State Med Soc. 2002;154(3):118-120.
  6. Zeitels SM, Casiano RR, Gardner GM, et al. Management of common voice problems: Committee report. Otolaryngol Head Neck Surg. 2002;126(4):333-348.
  7. Casper JK, Murry T. Voice therapy methods in dysphonia. Otolaryngol Clin North Am. 2000;33(5):983-1002.
  8. Samlan RA, Webster KT. Swallowing and speech therapy after definitive treatment for laryngeal cancer. Otolaryngol Clin North Am. 2002;35(5):1115-1133.
  9. Dejonckere PH. Clinical implementation of a multidimensional basic protocol for assessing functional results of voice therapy. A preliminary study. Rev Laryngol Otol Rhinol (Bord). 2000;121(5):311-313.
  10. Sama A, Carding PN, Price S, et al. The clinical features of functional dysphonia. Laryngoscope. 2001;111(3):458-463.
  11. Roy N. Functional dysphonia. Curr Opin Otolaryngol Head Neck Surg. 2003;11(3):144-148.
  12. Miller S. Voice therapy for vocal fold paralysis. Otolaryngol Clin North Am. 2004;37(1):105-119.
  13. Branski RC, Murray T. Voice therapy. eMedicine Otolaryngology and Facial Plastic Surgery Topic 683. Omaha, NE: eMedicine.com; updated August 4, 2005. Available at: http://www.emedicine.com/ent/topic683.htm. Accessed September 30, 2005.
  14. van Gogh CD, Verdonck-de Leeuw IM, Boon-Kamma BA, et al. The efficacy of voice therapy in patients after treatment for early glottic carcinoma. Cancer. 2006;106(1):95-105.
  15. Chiara T, Martin D, Sapienza C. Expiratory muscle strength training: Speech production outcomes in patients with multiple sclerosis. Neurorehabil Neural Repair. 2007;21(3):239-249.
  16. Ruotsalainen JH, Sellman J, Lehto L, et al. Interventions for treating functional dysphonia in adults. Cochrane Database Syst Rev. 2007;(3):CD006373.


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Copyright Aetna Inc. All rights reserved. Clinical Policy Bulletins are developed by Aetna to assist in administering plan benefits and constitute neither offers of coverage nor medical advice. This Clinical Policy Bulletin contains only a partial, general description of plan or program benefits and does not constitute a contract. Aetna does not provide health care services and, therefore, cannot guarantee any results or outcomes. Participating providers are independent contractors in private practice and are neither employees nor agents of Aetna or its affiliates. Treating providers are solely responsible for medical advice and treatment of members. This Clinical Policy Bulletin may be updated and therefore is subject to change.
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